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Rotein TCP-1 and the `chaperonin' family of bacterial (GroEL, 60-65 kDa heat shock antigen) and eukaryotic proteins. Biochem. Int. 20:833?41. 96. Gupta, R. S. 1995. Evolution of the chaperonin families (Hsp60, Hsp10 and Tcp-1) of proteins and the origin of eukaryotic cells. Mol. Microbiol. 15: 1?1. 97. Gupta, R. S. 1995. Phylogenetic analysis of the 90 kD heat shock family of protein sequences and
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Rotein TCP-1 and the `chaperonin' family of bacterial (GroEL, 60-65 kDa heat shock antigen) and eukaryotic proteins. Biochem. Int. 20:833?41. 96. Gupta, R. S. 1995. Evolution of the chaperonin families (Hsp60, Hsp10 and Tcp-1) of proteins and the origin of eukaryotic cells. Mol. Microbiol. 15: 1?1. 97. Gupta, R. S. 1995. Phylogenetic analysis of the 90 kD heat shock family of protein sequences and
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S and signature sequences: evolutionary relationships within prokaryotes and between prokaryotes and eukaryotes. Antonie Leeuwenhoek 72:49?1. 100. Gupta, R. S. 1998. Life's third domain (Archaea): an established fact or an endangered paradigm? A new proposal for classification of organisms based on protein sequences and cell structure. Theor. Popul. Biol. 54:91?104. 101. Gupta, R. S. 1998. What ar
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Imeric origin for the eukaryotic cell nucleus. Curr. Biol. 4:1104?114. 109. Hartman, H. 1984. The origin of the eukaryotic cell. Speculations Sci. Technol. 7:77?1. 110. Hasegawa, M., and M. Fujiwara. 1993. Relative efficiencies of the maximum likelihood, maximum parsimony, and neighbor-joining methods for estimating protein phylogeny. Mol. Phylogenet. Evol. 2:1?. 111. Hasegawa, M., and T. Hashimot
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Hựu vân, yếu đáo nhất tâm bất loạn cảnh giới, diệc vô tha thuật. Đây cũng là lời của đại sư Ngẫu Ích. Quý vị hy vọng niệm đến nhất tâm bất loạn, cũng không ngoài phương pháp này. Phương pháp đó là gì? Tối sơ hạ thủ, tu dụng số châu, ký đắc phân minh, khắc định khóa trình, quyết định vô khuyết, cửu cửu thuần thục, bất niệm tự niệm. Nhiên hậu ký số diệc đắc, bất ký sô diệc đắc. Tổ sư khuyên chúng ta
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Hựu vân, yếu đáo nhất tâm bất loạn cảnh giới, diệc vô tha thuật. Đây cũng là lời của đại sư Ngẫu Ích. Quý vị hy vọng niệm đến nhất tâm bất loạn, cũng không ngoài phương pháp này. Phương pháp đó là gì? Tối sơ hạ thủ, tu dụng số châu, ký đắc phân minh, khắc định khóa trình, quyết định vô khuyết, cửu cửu thuần thục, bất niệm tự niệm. Nhiên hậu ký số diệc đắc, bất ký sô diệc đắc. Tổ sư khuyên chúng ta
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Tứ thánh pháp giới trong mười pháp giới là tịnh độ. Lục đạo trong cõi phương tiện hữu dư là uế độ. Cõi đồng cư trong thế giới tây phương Cực lạc là tịnh độ. Điều này rất khó. Vì sao cõi đồng cư của thế giới Cực Lạc đều là thanh tịnh trang nghiêm? Vì phàm sanh đến thế giới Cực Lạc đều là chư thượng thiện nhơn. Nói cách khác, đều là đầy đủ điều kiện thượng thiện, mới có thể đến đây. Đây là sự thù th
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In this research, the effect regarding roxithromycin (ROX) on unstable essential fatty acid (VFA) recuperation coming from WAS anaerobic fermentation had been looked into. The particular trial and error results showed #links# in which ROX developed a good factor on the creation of VFAs. With all the improve of ROX dosages from Zero to 100?mg/kg TSS, the maximum build up regarding VFAs increased fr
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In these studies, the effects regarding roxithromycin (ROX) in volatile fatty acid (VFA) recovery coming from Ended up being anaerobic fermentation ended up being looked at. Your fresh outcomes confirmed #links# that ROX designed a good share on the creation of VFAs. With all the increase associated with ROX doses from 2 to be able to 100?mg/kg TSS, the maximum deposition involving VFAs increased
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Ransfection assays [11]. To determine if these proteins, together with BORIS, can form secondary complexes with each other in the absence of DNA and to understand better the mechanism underlying the regulation of MAGE-A1 expression, we carried out in vitro proteinprotein interaction assays. Each of the proteins was either a resin-bound "bait" fusion protein or a [35S]-L-methionine labeled "prey" p
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Noma in situ; DC-SCRIPT, dendritic cell-specific transcript gene; ERBB2+, HER2neu-positive; ESR, estrogen receptor gene; IDC, infiltrating ductal carcinoma; ILC, infiltrating lobular carcinoma; PGR, progesterone receptor gene; pT1, small tumor without lymphatic/vascular invasion.(Spearman's rho = 0.87; P
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Lyzed its impact on the modifications of histones bound at the MAGE-A1 promoters. To investigate the changes in the histone signature of MAGE-A1 promoter, it was compared in basal MCF-7 cells (no expression of MAGE-A1, Table 1) to the signature in MCF-7 cells stimulated by 5-aza-CdR with/without TSA or transfected with the expression plasmid encoding for BORIS. For these analyses we used antibodie
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Ith DFS, MFS, OS, and PFS, respectively. In multivariable analysis, Cox proportional hazards models for DFS, MFS, OS, and PFS were applied to test DCSCRIPT levels added to models with traditional factors. The proportional hazards assumptions were checked with Schoenfeld residuals. The analyses were stratified if necessary. The models for DFS, MFS, and OS for LNN patients who had not received adjuv
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Ic immunoprecipitation and input DNA. H3K9, Lysine 9 of histone H3; H4K8, Lysine 8 of histone H4; H3K4, Lysine 4 of histone H3; H4K20, Lysine 20 of histone H4; ac, acetylated; me, monomethylated; me2, dimethylated; me3, trimethylated. The significant p-values are shown.Schwarzenbach et al. BMC Cancer 2014, 14:796 http://www.biomedcentral.com/1471-2407/14/Page 10 ofTherefore, the predominant occurr
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Ms into six subfamilies (sf1 to sf6) based on their unique 3' terminal sequences, which were used to design 6 Taqman probes for quantitative real-time PCR [17]. With the exception of sf1, which detects the original BORIS form (B0) that was transfected into the cell lines, we measured the relative units of BORIS isoforms sf2 to sf6 in basal and BORIS-transfected MCF-7 and BCM1 cells. As shown in Fi
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Ith DFS, MFS, OS, and PFS, respectively. In multivariable analysis, Cox proportional hazards models for DFS, MFS, OS, and PFS were applied to test DCSCRIPT levels added to models with traditional factors. The proportional hazards assumptions were checked with Schoenfeld residuals. The analyses were stratified if necessary. The models for DFS, MFS, and OS for LNN patients who had not received adjuv
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Me PCR conditions for these SYBR-based assays were as described previously [16,17]. Forty rounds of amplification were performed, and fluorescent signals of the Taqman probe or SYBR green signal were used to generate cycle threshold (Ct) values from which mRNA expression levels were calculated. Ct values of HPRT1 and B2M were adjusted to the higher HMBS Ct values. Next, the expression levels of DC
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